The structure-carrying and form-determining part of the human erythrocyte-membrane [membrane of the red blood cells] consists of a micellar protein frame - (true secondary extra- or mesomolecular organ-membrane, in contrast to the functional intramolecular primary denaturation membrane of the cytoplasm [= plasm of the cell content except the nucleus] ), - a micellar protein frame filled up with lipoids regulating permeability [of the membrane]. A malaria plasmodium, penetrating into an erythrocyte, extracts for own use the lipoids from the membrane and only leaves behind the protein skeleton of the erythrocite membrane and own metabolites. During the penetration of the merocyte (sporulation product) into the new host cell, it removes its own lipoid membrane [so it crawls out of its own skin, or shell] and grows, to begin with, without a shell of its own. "As a result of the demonstration of a (organ-like?) membrane, a secure morphological distinction of bacteria from certain virusses which never have any membrane, has become possible" (Bergold). Here [I presume in virusses] it is, probably, only the simple surface-of-solids-orientation that is present. "In artificially produced protein membranes (films on water) one has observed that they are penetrable to lipoids [i.e. lipoids can cross them] (compare intracellular exchange). It is also remarkable that those substances which become adsorbed to protein-films, coagulate red blood cells, whereas those substances which penetrate into protein-films cause hemolysis of erythrocites" [i.e. disintegration of red blood cells].